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Griswold DE, Webb EF, Breton J, White JR, Marshall PJ, Torphy TJ (1993). „Effect of selective phosphodiesterase type IV inhibitor, rolipram, on fluid and cellular phases of inflammatory response”. Inflammation. 17 (3): 333—44. PMID7687237. doi:10.1007/BF00918994.
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Wachtel H. (1983). „Potential antidepressant activity of rolipram and other selective cyclic adenosine 3',5'-monophosphate phosphodiesterase inhibitors”. Neuropharmacology. 22 (3): 267—72. PMID6302550. doi:10.1016/0028-3908(83)90239-3.
Maxwell CR, Kanes SJ, Abel T, Siegel SJ (2004). „Phosphodiesterase inhibitors: a novel mechanism for receptor-independent antipsychotic medications”. Neuroscience. 129 (1): 101—7. PMID15489033. doi:10.1016/j.neuroscience.2004.07.038.
Kanes SJ, Tokarczyk J, Siegel SJ, Bilker W, Abel T, Kelly MP (2006). „Rolipram: A specific phosphodiesterase 4 inhibitor with potential antipsychotic activity”. Neuroscience. 144 (1): 239—46. PMID17081698. doi:10.1016/j.neuroscience.2006.09.026.
Lelkes Z, Alföldi P, Erdos A, Benedek G (1998). „Rolipram, an antidepressant that increases the availability of cAMP, transiently enhances wakefulness in rats”. Pharmacology, Biochemistry and Behaviour. 60 (4): 835—9. PMID9700966. doi:10.1016/S0091-3057(98)00038-0.
Chen RW, Williams AJ, Liao Z, Yao C, Tortella FC, Dave JR (2007). „Broad spectrum neuroprotection profile of phosphodiesterase inhibitors as related to modulation of cell-cycle elements and caspase-3 activation”. Neuroscience Letters. 418 (2): 165—9. PMID17398001. doi:10.1016/j.neulet.2007.03.033.
MF, Beal; C, Cleren; et al. (2005). „Oxidative Damage in Parkinson's Disease”. U.S. Army Medical Research and Materiel CommandFort Detrick, Maryland 21702-5012. Архивирано из оригинала 23. 05. 2012. г. Приступљено 13. 11. 2009.