Ma, Juan; Zhang Guang-Yi (2003). „Lithium reduced N-methyl-D-aspartate receptor subunit 2A tyrosine phosphorylation and its interactions with Src and Fyn mediated by PSD-95 in rat hippocampus following cerebral ischemia”. Neurosci. Lett. Ireland. 348 (3): 185—9. ISSN0304-3940. PMID12932824. doi:10.1016/S0304-3940(03)00784-5.
Takagi, N; et al. (1999). „The effect of transient global ischemia on the interaction of Src and Fyn with the N-methyl-D-aspartate receptor and postsynaptic densities: possible involvement of Src homology 2 domains”. J. Cereb. Blood Flow Metab. UNITED STATES. 19 (8): 880—8. ISSN0271-678X. PMID10458595. doi:10.1097/00004647-199908000-00007.
Bourguignon, L Y; et al. (2001). „CD44 interaction with c-Src kinase promotes cortactin-mediated cytoskeleton function and hyaluronic acid-dependent ovarian tumor cell migration”. J. Biol. Chem. United States. 276 (10): 7327—36. ISSN0021-9258. PMID11084024. doi:10.1074/jbc.M006498200.
Wan, Kah Fei; et al. (2003). „The inhibitory gamma subunit of the type 6 retinal cGMP phosphodiesterase functions to link c-Src and G-protein-coupled receptor kinase 2 in a signaling unit that regulates p42/p44 mitogen-activated protein kinase by epidermal growth factor”. J. Biol. Chem. United States. 278 (20): 18658—63. ISSN0021-9258. PMID12624098. doi:10.1074/jbc.M212103200.
Olayioye, M A; et al. (1999). „ErbB receptor-induced activation of stat transcription factors is mediated by Src tyrosine kinases”. J. Biol. Chem. UNITED STATES. 274 (24): 17209—18. ISSN0021-9258. PMID10358079. doi:10.1074/jbc.274.24.17209.
Cirri, P; et al. (1997). „c-Src activates both STAT1 and STAT3 in PDGF-stimulated NIH3T3 cells”. Biochem. Biophys. Res. Commun. UNITED STATES. 239 (2): 493—7. ISSN0006-291X. PMID9344858. doi:10.1006/bbrc.1997.7493.
Zisch, A H; et al. (1998). „Complex formation between EphB2 and Src requires phosphorylation of tyrosine 611 in the EphB2 juxtamembrane region”. Oncogene. ENGLAND. 16 (20): 2657—70. ISSN0950-9232. PMID9632142. doi:10.1038/sj.onc.1201823.
Zisch, A H (2000). „Replacing two conserved tyrosines of the EphB2 receptor with glutamic acid prevents binding of SH2 domains without abrogating kinase activity and biological responses”. Oncogene. ENGLAND. 19 (2): 177—87. ISSN0950-9232. PMID10644995. doi:10.1038/sj.onc.1203304.
Unni, Emmanual; et al. (2004). „Changes in androgen receptor nongenotropic signaling correlate with transition of LNCaP cells to androgen independence”. Cancer Res. United States. 64 (19): 7156—68. ISSN0008-5472. PMID15466214. doi:10.1158/0008-5472.CAN-04-1121.
Powell, S M; et al. (2004). „Mechanisms of androgen receptor signalling via steroid receptor coactivator-1 in prostate”. Endocr. Relat. Cancer. England. 11 (1): 117—30. ISSN1351-0088. PMID15027889. doi:10.1677/erc.0.0110117.
Seibenhener, M L; et al. (1999). „Identification of Src as a novel atypical protein kinase C-interacting protein”. Mol. Cell Biol. Res. Commun. UNITED STATES. 2 (1): 28—31. ISSN1522-4724. PMID10527887. doi:10.1006/mcbr.1999.0140.
Cao, W; et al. (2000). „Direct binding of activated c-Src to the beta 3-adrenergic receptor is required for MAP kinase activation”. J. Biol. Chem. UNITED STATES. 275 (49): 38131—4. ISSN0021-9258. PMID11013230. doi:10.1074/jbc.C000592200.
Keely, S J; et al. (2000). „Carbachol-stimulated transactivation of epidermal growth factor receptor and mitogen-activated protein kinase in T(84) cells is mediated by intracellular ca(2+), PYK-2, and p60(src)”. J. Biol. Chem. UNITED STATES. 275 (17): 12619—25. ISSN0021-9258. PMID10777553. doi:10.1074/jbc.275.17.12619.
Sato, K; et al. (2000). „Adaptor protein Shc undergoes translocation and mediates up-regulation of the tyrosine kinase c-Src in EGF-stimulated A431 cells”. Genes Cells. ENGLAND. 5 (9): 749—64. ISSN1356-9597. PMID10971656. doi:10.1046/j.1365-2443.2000.00358.x.
Messina, Samantha; et al. (2003). „Specific interactions of neuronal focal adhesion kinase isoforms with Src kinases and amphiphysin”. J. Neurochem. England. 84 (2): 253—65. ISSN0022-3042. PMID12558988. doi:10.1046/j.1471-4159.2003.01519.x.
Lebrun, P; et al. (1998). „Insulin receptor substrate-1 as a signaling molecule for focal adhesion kinase pp. 125(FAK) and pp. 60(src)”. J. Biol. Chem. UNITED STATES. 273 (48): 32244—53. ISSN0021-9258. PMID9822703. doi:10.1074/jbc.273.48.32244.
Zhou, Jian; et al. (2003). „Characterization of a novel negative regulator (DOC-2/DAB2) of c-Src in normal prostatic epithelium and cancer”. J. Biol. Chem. United States. 278 (9): 6936—41. ISSN0021-9258. PMID12473651. doi:10.1074/jbc.M210628200.
Sotgia, F; et al. (2001). „Tyrosine phosphorylation of beta-dystroglycan at its WW domain binding motif, PPxY, recruits SH2 domain containing proteins”. Biochemistry. United States. 40 (48): 14585—92. ISSN0006-2960. PMID11724572. doi:10.1021/bi011247r.
Slentz-Kesler, K; et al. (2000). „Identification of the human Mnk2 gene (MKNK2) through protein interaction with estrogen receptor beta”. Genomics. UNITED STATES. 69 (1): 63—71. ISSN0888-7543. PMID11013076. doi:10.1006/geno.2000.6299.
Soutoglou, E; et al. (2000). „Transcriptional activation by hepatocyte nuclear factor-1 requires synergism between multiple coactivator proteins”. J. Biol. Chem. UNITED STATES. 275 (17): 12515—20. ISSN0021-9258. PMID10777539. doi:10.1074/jbc.275.17.12515.
Stelzl, Ulrich; et al. (2005). „A human protein-protein interaction network: a resource for annotating the proteome”. Cell. United States. 122 (6): 957—68. ISSN0092-8674. PMID16169070. doi:10.1016/j.cell.2005.08.029.
Oneyama, Chitose; et al. (2002). „UCS15A, a novel small molecule, SH3 domain-mediated protein-protein interaction blocking drug”. Oncogene. England. 21 (13): 2037—50. ISSN0950-9232. PMID11960376. doi:10.1038/sj.onc.1205271.
Li, Y; et al. (2001). „The c-Src tyrosine kinase regulates signaling of the human DF3/MUC1 carcinoma-associated antigen with GSK3 beta and beta-catenin”. J. Biol. Chem. United States. 276 (9): 6061—4. ISSN0021-9258. PMID11152665. doi:10.1074/jbc.C000754200.
Li, Y; et al. (2001). „The epidermal growth factor receptor regulates interaction of the human DF3/MUC1 carcinoma antigen with c-Src and beta-catenin”. J. Biol. Chem. United States. 276 (38): 35239—42. ISSN0021-9258. PMID11483589. doi:10.1074/jbc.C100359200.
Kumar, S; et al. (1999). „Negative regulation of PYK2/related adhesion focal tyrosine kinase signal transduction by hematopoietic tyrosine phosphatase SHPTP1”. J. Biol. Chem. UNITED STATES. 274 (43): 30657—63. ISSN0021-9258. PMID10521452. doi:10.1074/jbc.274.43.30657.
Dikic, I; et al. (1996). „A role for Pyk2 and Src in linking G-protein-coupled receptors with MAP kinase activation”. Nature. ENGLAND. 383 (6600): 547—50. ISSN0028-0836. PMID8849729. doi:10.1038/383547a0.
Burnham, M R; et al. (1999). „The role of SRC-CAS interactions in cellular transformation: ectopic expression of the carboxy terminus of CAS inhibits SRC-CAS interaction but has no effect on cellular transformation”. Mol. Carcinog. UNITED STATES. 26 (1): 20—31. ISSN0899-1987. PMID10487518. doi:10.1002/(SICI)1098-2744(199909)26:1<20::AID-MC3>3.0.CO;2-M.
Wei, Lin; et al. (2002). „Anchorage-independent phosphorylation of p130(Cas) protects lung adenocarcinoma cells from anoikis”. J. Cell. Biochem. United States. 87 (4): 439—49. ISSN0730-2312. PMID12397603. doi:10.1002/jcb.10322.
Donaldson, J C; et al. (2000). „Crk-associated substrate p130(Cas) interacts with nephrocystin and both proteins localize to cell-cell contacts of polarized epithelial cells”. Exp. Cell Res. UNITED STATES. 256 (1): 168—78. ISSN0014-4827. PMID10739664. doi:10.1006/excr.2000.4822.
Kim, H J; et al. (1998). „Steroid receptor coactivator-1 interacts with serum response factor and coactivates serum response element-mediated transactivations”. J. Biol. Chem. UNITED STATES. 273 (44): 28564—7. ISSN0021-9258. PMID9786846. doi:10.1074/jbc.273.44.28564.
Lee, S K; et al. (1999). „A nuclear factor, ASC-2, as a cancer-amplified transcriptional coactivator essential for ligand-dependent transactivation by nuclear receptors in vivo”. J. Biol. Chem. UNITED STATES. 274 (48): 34283—93. ISSN0021-9258. PMID10567404. doi:10.1074/jbc.274.48.34283.
Lee, S K; et al. (2000). „Activating protein-1, nuclear factor-kappaB, and serum response factor as novel target molecules of the cancer-amplified transcription coactivator ASC-2”. Mol. Endocrinol. UNITED STATES. 14 (6): 915—25. ISSN0888-8809. PMID10847592. doi:10.1210/me.14.6.915.
Lee, S K; et al. (2001). „Two distinct nuclear receptor-interaction domains and CREB-binding protein-dependent transactivation function of activating signal cointegrator-2”. Mol. Endocrinol. United States. 15 (2): 241—54. ISSN0888-8809. PMID11158331. doi:10.1210/me.15.2.241.
Banin, S; et al. (1996). „Wiskott-Aldrich syndrome protein (WASp) is a binding partner for c-Src family protein-tyrosine kinases”. Curr. Biol. ENGLAND. 6 (8): 981—8. ISSN0960-9822. PMID8805332. doi:10.1016/S0960-9822(02)00642-5.
Finan, P M; et al. (1996). „Identification of regions of the Wiskott-Aldrich syndrome protein responsible for association with selected Src homology 3 domains”. J. Biol. Chem. UNITED STATES. 271 (42): 26291—5. ISSN0021-9258. PMID8824280. doi:10.1074/jbc.271.42.26291.
Moon, Sun Young; et al. (2003). „Characterization of a brain-specific Rho GTPase-activating protein, p200RhoGAP”. J. Biol. Chem. United States. 278 (6): 4151—9. ISSN0021-9258. PMID12454018. doi:10.1074/jbc.M207789200.
Giglione, C; et al. (2001). „Differential actions of p60c-Src and Lck kinases on the Ras regulators p120-GAP and GDP/GTP exchange factor CDC25Mm”. Eur. J. Biochem. Germany. 268 (11): 3275—83. ISSN0014-2956. PMID11389730. doi:10.1046/j.1432-1327.2001.02230.x.
Ma, Juan; Zhang Guang-Yi (2003). „Lithium reduced N-methyl-D-aspartate receptor subunit 2A tyrosine phosphorylation and its interactions with Src and Fyn mediated by PSD-95 in rat hippocampus following cerebral ischemia”. Neurosci. Lett. Ireland. 348 (3): 185—9. ISSN0304-3940. PMID12932824. doi:10.1016/S0304-3940(03)00784-5.
Takagi, N; et al. (1999). „The effect of transient global ischemia on the interaction of Src and Fyn with the N-methyl-D-aspartate receptor and postsynaptic densities: possible involvement of Src homology 2 domains”. J. Cereb. Blood Flow Metab. UNITED STATES. 19 (8): 880—8. ISSN0271-678X. PMID10458595. doi:10.1097/00004647-199908000-00007.
Cleghon, V; Morrison D K (1994). „Raf-1 interacts with Fyn and Src in a non-phosphotyrosine-dependent manner”. J. Biol. Chem. UNITED STATES. 269 (26): 17749—55. ISSN0021-9258. PMID7517401.
Bourguignon, L Y; et al. (2001). „CD44 interaction with c-Src kinase promotes cortactin-mediated cytoskeleton function and hyaluronic acid-dependent ovarian tumor cell migration”. J. Biol. Chem. United States. 276 (10): 7327—36. ISSN0021-9258. PMID11084024. doi:10.1074/jbc.M006498200.
Wan, Kah Fei; et al. (2003). „The inhibitory gamma subunit of the type 6 retinal cGMP phosphodiesterase functions to link c-Src and G-protein-coupled receptor kinase 2 in a signaling unit that regulates p42/p44 mitogen-activated protein kinase by epidermal growth factor”. J. Biol. Chem. United States. 278 (20): 18658—63. ISSN0021-9258. PMID12624098. doi:10.1074/jbc.M212103200.
Olayioye, M A; et al. (1999). „ErbB receptor-induced activation of stat transcription factors is mediated by Src tyrosine kinases”. J. Biol. Chem. UNITED STATES. 274 (24): 17209—18. ISSN0021-9258. PMID10358079. doi:10.1074/jbc.274.24.17209.
Cirri, P; et al. (1997). „c-Src activates both STAT1 and STAT3 in PDGF-stimulated NIH3T3 cells”. Biochem. Biophys. Res. Commun. UNITED STATES. 239 (2): 493—7. ISSN0006-291X. PMID9344858. doi:10.1006/bbrc.1997.7493.
Zisch, A H; et al. (1998). „Complex formation between EphB2 and Src requires phosphorylation of tyrosine 611 in the EphB2 juxtamembrane region”. Oncogene. ENGLAND. 16 (20): 2657—70. ISSN0950-9232. PMID9632142. doi:10.1038/sj.onc.1201823.
Zisch, A H (2000). „Replacing two conserved tyrosines of the EphB2 receptor with glutamic acid prevents binding of SH2 domains without abrogating kinase activity and biological responses”. Oncogene. ENGLAND. 19 (2): 177—87. ISSN0950-9232. PMID10644995. doi:10.1038/sj.onc.1203304.
Unni, Emmanual; et al. (2004). „Changes in androgen receptor nongenotropic signaling correlate with transition of LNCaP cells to androgen independence”. Cancer Res. United States. 64 (19): 7156—68. ISSN0008-5472. PMID15466214. doi:10.1158/0008-5472.CAN-04-1121.
Powell, S M; et al. (2004). „Mechanisms of androgen receptor signalling via steroid receptor coactivator-1 in prostate”. Endocr. Relat. Cancer. England. 11 (1): 117—30. ISSN1351-0088. PMID15027889. doi:10.1677/erc.0.0110117.
Seibenhener, M L; et al. (1999). „Identification of Src as a novel atypical protein kinase C-interacting protein”. Mol. Cell Biol. Res. Commun. UNITED STATES. 2 (1): 28—31. ISSN1522-4724. PMID10527887. doi:10.1006/mcbr.1999.0140.
Cao, W; et al. (2000). „Direct binding of activated c-Src to the beta 3-adrenergic receptor is required for MAP kinase activation”. J. Biol. Chem. UNITED STATES. 275 (49): 38131—4. ISSN0021-9258. PMID11013230. doi:10.1074/jbc.C000592200.
Keely, S J; et al. (2000). „Carbachol-stimulated transactivation of epidermal growth factor receptor and mitogen-activated protein kinase in T(84) cells is mediated by intracellular ca(2+), PYK-2, and p60(src)”. J. Biol. Chem. UNITED STATES. 275 (17): 12619—25. ISSN0021-9258. PMID10777553. doi:10.1074/jbc.275.17.12619.
Sato, K; et al. (2000). „Adaptor protein Shc undergoes translocation and mediates up-regulation of the tyrosine kinase c-Src in EGF-stimulated A431 cells”. Genes Cells. ENGLAND. 5 (9): 749—64. ISSN1356-9597. PMID10971656. doi:10.1046/j.1365-2443.2000.00358.x.
Hecker, Timothy P; et al. (2002). „Focal adhesion kinase enhances signaling through the Shc/extracellular signal-regulated kinase pathway in anaplastic astrocytoma tumor biopsy samples”. Cancer Res. United States. 62 (9): 2699—707. ISSN0008-5472. PMID11980671.
Messina, Samantha; et al. (2003). „Specific interactions of neuronal focal adhesion kinase isoforms with Src kinases and amphiphysin”. J. Neurochem. England. 84 (2): 253—65. ISSN0022-3042. PMID12558988. doi:10.1046/j.1471-4159.2003.01519.x.
Lebrun, P; et al. (1998). „Insulin receptor substrate-1 as a signaling molecule for focal adhesion kinase pp. 125(FAK) and pp. 60(src)”. J. Biol. Chem. UNITED STATES. 273 (48): 32244—53. ISSN0021-9258. PMID9822703. doi:10.1074/jbc.273.48.32244.
Zhou, Jian; et al. (2003). „Characterization of a novel negative regulator (DOC-2/DAB2) of c-Src in normal prostatic epithelium and cancer”. J. Biol. Chem. United States. 278 (9): 6936—41. ISSN0021-9258. PMID12473651. doi:10.1074/jbc.M210628200.
Sotgia, F; et al. (2001). „Tyrosine phosphorylation of beta-dystroglycan at its WW domain binding motif, PPxY, recruits SH2 domain containing proteins”. Biochemistry. United States. 40 (48): 14585—92. ISSN0006-2960. PMID11724572. doi:10.1021/bi011247r.
Slentz-Kesler, K; et al. (2000). „Identification of the human Mnk2 gene (MKNK2) through protein interaction with estrogen receptor beta”. Genomics. UNITED STATES. 69 (1): 63—71. ISSN0888-7543. PMID11013076. doi:10.1006/geno.2000.6299.
Soutoglou, E; et al. (2000). „Transcriptional activation by hepatocyte nuclear factor-1 requires synergism between multiple coactivator proteins”. J. Biol. Chem. UNITED STATES. 275 (17): 12515—20. ISSN0021-9258. PMID10777539. doi:10.1074/jbc.275.17.12515.
Stelzl, Ulrich; et al. (2005). „A human protein-protein interaction network: a resource for annotating the proteome”. Cell. United States. 122 (6): 957—68. ISSN0092-8674. PMID16169070. doi:10.1016/j.cell.2005.08.029.
Oneyama, Chitose; et al. (2002). „UCS15A, a novel small molecule, SH3 domain-mediated protein-protein interaction blocking drug”. Oncogene. England. 21 (13): 2037—50. ISSN0950-9232. PMID11960376. doi:10.1038/sj.onc.1205271.
Li, Y; et al. (2001). „The c-Src tyrosine kinase regulates signaling of the human DF3/MUC1 carcinoma-associated antigen with GSK3 beta and beta-catenin”. J. Biol. Chem. United States. 276 (9): 6061—4. ISSN0021-9258. PMID11152665. doi:10.1074/jbc.C000754200.
Li, Y; et al. (2001). „The epidermal growth factor receptor regulates interaction of the human DF3/MUC1 carcinoma antigen with c-Src and beta-catenin”. J. Biol. Chem. United States. 276 (38): 35239—42. ISSN0021-9258. PMID11483589. doi:10.1074/jbc.C100359200.
Kumar, S; et al. (1999). „Negative regulation of PYK2/related adhesion focal tyrosine kinase signal transduction by hematopoietic tyrosine phosphatase SHPTP1”. J. Biol. Chem. UNITED STATES. 274 (43): 30657—63. ISSN0021-9258. PMID10521452. doi:10.1074/jbc.274.43.30657.
Dikic, I; et al. (1996). „A role for Pyk2 and Src in linking G-protein-coupled receptors with MAP kinase activation”. Nature. ENGLAND. 383 (6600): 547—50. ISSN0028-0836. PMID8849729. doi:10.1038/383547a0.
Burnham, M R; et al. (1999). „The role of SRC-CAS interactions in cellular transformation: ectopic expression of the carboxy terminus of CAS inhibits SRC-CAS interaction but has no effect on cellular transformation”. Mol. Carcinog. UNITED STATES. 26 (1): 20—31. ISSN0899-1987. PMID10487518. doi:10.1002/(SICI)1098-2744(199909)26:1<20::AID-MC3>3.0.CO;2-M.
Wei, Lin; et al. (2002). „Anchorage-independent phosphorylation of p130(Cas) protects lung adenocarcinoma cells from anoikis”. J. Cell. Biochem. United States. 87 (4): 439—49. ISSN0730-2312. PMID12397603. doi:10.1002/jcb.10322.
Donaldson, J C; et al. (2000). „Crk-associated substrate p130(Cas) interacts with nephrocystin and both proteins localize to cell-cell contacts of polarized epithelial cells”. Exp. Cell Res. UNITED STATES. 256 (1): 168—78. ISSN0014-4827. PMID10739664. doi:10.1006/excr.2000.4822.
Karlsson, T; et al. (1995). „Molecular interactions of the Src homology 2 domain protein Shb with phosphotyrosine residues, tyrosine kinase receptors and Src homology 3 domain proteins”. Oncogene. ENGLAND. 10 (8): 1475—83. ISSN0950-9232. PMID7537362.
Kim, H J; et al. (1998). „Steroid receptor coactivator-1 interacts with serum response factor and coactivates serum response element-mediated transactivations”. J. Biol. Chem. UNITED STATES. 273 (44): 28564—7. ISSN0021-9258. PMID9786846. doi:10.1074/jbc.273.44.28564.
Lee, S K; et al. (1999). „A nuclear factor, ASC-2, as a cancer-amplified transcriptional coactivator essential for ligand-dependent transactivation by nuclear receptors in vivo”. J. Biol. Chem. UNITED STATES. 274 (48): 34283—93. ISSN0021-9258. PMID10567404. doi:10.1074/jbc.274.48.34283.
Lee, S K; et al. (2000). „Activating protein-1, nuclear factor-kappaB, and serum response factor as novel target molecules of the cancer-amplified transcription coactivator ASC-2”. Mol. Endocrinol. UNITED STATES. 14 (6): 915—25. ISSN0888-8809. PMID10847592. doi:10.1210/me.14.6.915.
Lee, S K; et al. (2001). „Two distinct nuclear receptor-interaction domains and CREB-binding protein-dependent transactivation function of activating signal cointegrator-2”. Mol. Endocrinol. United States. 15 (2): 241—54. ISSN0888-8809. PMID11158331. doi:10.1210/me.15.2.241.
Banin, S; et al. (1996). „Wiskott-Aldrich syndrome protein (WASp) is a binding partner for c-Src family protein-tyrosine kinases”. Curr. Biol. ENGLAND. 6 (8): 981—8. ISSN0960-9822. PMID8805332. doi:10.1016/S0960-9822(02)00642-5.
Finan, P M; et al. (1996). „Identification of regions of the Wiskott-Aldrich syndrome protein responsible for association with selected Src homology 3 domains”. J. Biol. Chem. UNITED STATES. 271 (42): 26291—5. ISSN0021-9258. PMID8824280. doi:10.1074/jbc.271.42.26291.
Moon, Sun Young; et al. (2003). „Characterization of a brain-specific Rho GTPase-activating protein, p200RhoGAP”. J. Biol. Chem. United States. 278 (6): 4151—9. ISSN0021-9258. PMID12454018. doi:10.1074/jbc.M207789200.
Giglione, C; et al. (2001). „Differential actions of p60c-Src and Lck kinases on the Ras regulators p120-GAP and GDP/GTP exchange factor CDC25Mm”. Eur. J. Biochem. Germany. 268 (11): 3275—83. ISSN0014-2956. PMID11389730. doi:10.1046/j.1432-1327.2001.02230.x.
Ma, Juan; Zhang Guang-Yi (2003). „Lithium reduced N-methyl-D-aspartate receptor subunit 2A tyrosine phosphorylation and its interactions with Src and Fyn mediated by PSD-95 in rat hippocampus following cerebral ischemia”. Neurosci. Lett. Ireland. 348 (3): 185—9. ISSN0304-3940. PMID12932824. doi:10.1016/S0304-3940(03)00784-5.
Takagi, N; et al. (1999). „The effect of transient global ischemia on the interaction of Src and Fyn with the N-methyl-D-aspartate receptor and postsynaptic densities: possible involvement of Src homology 2 domains”. J. Cereb. Blood Flow Metab. UNITED STATES. 19 (8): 880—8. ISSN0271-678X. PMID10458595. doi:10.1097/00004647-199908000-00007.
Cleghon, V; Morrison D K (1994). „Raf-1 interacts with Fyn and Src in a non-phosphotyrosine-dependent manner”. J. Biol. Chem. UNITED STATES. 269 (26): 17749—55. ISSN0021-9258. PMID7517401.
Bourguignon, L Y; et al. (2001). „CD44 interaction with c-Src kinase promotes cortactin-mediated cytoskeleton function and hyaluronic acid-dependent ovarian tumor cell migration”. J. Biol. Chem. United States. 276 (10): 7327—36. ISSN0021-9258. PMID11084024. doi:10.1074/jbc.M006498200.
Wan, Kah Fei; et al. (2003). „The inhibitory gamma subunit of the type 6 retinal cGMP phosphodiesterase functions to link c-Src and G-protein-coupled receptor kinase 2 in a signaling unit that regulates p42/p44 mitogen-activated protein kinase by epidermal growth factor”. J. Biol. Chem. United States. 278 (20): 18658—63. ISSN0021-9258. PMID12624098. doi:10.1074/jbc.M212103200.
Olayioye, M A; et al. (1999). „ErbB receptor-induced activation of stat transcription factors is mediated by Src tyrosine kinases”. J. Biol. Chem. UNITED STATES. 274 (24): 17209—18. ISSN0021-9258. PMID10358079. doi:10.1074/jbc.274.24.17209.
Cirri, P; et al. (1997). „c-Src activates both STAT1 and STAT3 in PDGF-stimulated NIH3T3 cells”. Biochem. Biophys. Res. Commun. UNITED STATES. 239 (2): 493—7. ISSN0006-291X. PMID9344858. doi:10.1006/bbrc.1997.7493.
Zisch, A H; et al. (1998). „Complex formation between EphB2 and Src requires phosphorylation of tyrosine 611 in the EphB2 juxtamembrane region”. Oncogene. ENGLAND. 16 (20): 2657—70. ISSN0950-9232. PMID9632142. doi:10.1038/sj.onc.1201823.
Zisch, A H (2000). „Replacing two conserved tyrosines of the EphB2 receptor with glutamic acid prevents binding of SH2 domains without abrogating kinase activity and biological responses”. Oncogene. ENGLAND. 19 (2): 177—87. ISSN0950-9232. PMID10644995. doi:10.1038/sj.onc.1203304.
Unni, Emmanual; et al. (2004). „Changes in androgen receptor nongenotropic signaling correlate with transition of LNCaP cells to androgen independence”. Cancer Res. United States. 64 (19): 7156—68. ISSN0008-5472. PMID15466214. doi:10.1158/0008-5472.CAN-04-1121.
Powell, S M; et al. (2004). „Mechanisms of androgen receptor signalling via steroid receptor coactivator-1 in prostate”. Endocr. Relat. Cancer. England. 11 (1): 117—30. ISSN1351-0088. PMID15027889. doi:10.1677/erc.0.0110117.
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