Церебрална амилоидна ангиопатија (Serbian Wikipedia)

Analysis of information sources in references of the Wikipedia article "Церебрална амилоидна ангиопатија" in Serbian language version.

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18nih.gov

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18doi.org

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2worldcat.org

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1harvard.edu

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1archive.org

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archive.org

Zannis V.I., Breslow J.L., Utermann G.; et al. (1982). „Proposed nomenclature of apoE isoproteins, apoE genotypes, and phenotypes”. J Lipid Res. 23 (6): 911—914. PMID 7130859. doi:10.1016/S0022-2275(20)38094-9. .

doi.org

Greenberg, S. M.; Charidimou, A. (2018). „Diagnosis of cerebral amyloid angiopathy: evolution of the Boston criteria”. Stroke. 49 (2): 491—497. PMC 5892842 . PMID 29335334. doi:10.1161/STROKEAHA.117.016990.
Revesz T., Holton J.L., Lashley T.; et al. (2009). „Genetics and molecular pathogenesis of sporadic and hereditary cerebral amyloid angiopathies”. Acta Neuropathol. 118 (1): 115—130. PMC 2844092 . PMID 19225789. doi:10.1007/s00401-009-0501-8. :115–130.
Roher A.E., Lowenson J.D., Clarke S.; et al. (1993). „beta-Amyloid-(1-42) is a major component of cerebrovascular amyloid deposits: implications for the pathology of Alzheimer disease”. Proc Natl Acad Sci U S A. 90 (22): 10836—10840. Bibcode:1993PNAS...9010836R. PMC 47873 . PMID 8248178. doi:10.1073/pnas.90.22.10836 . :10836–10840.
Attems J., Lintner F., Jellinger K.A. (2004). „Amyloid β peptide 1–42 highly correlates with capillary cerebral amyloid angiopathy and Alzheimer disease pathology”. Acta Neuropathol. 107 (5): 479—480. PMID 14986026. doi:10.1007/s00401-004-0866-7. :283–291.
Keable A., Fenna K., Yuen H.M.; et al. (2016). „Deposition of amyloid β in the walls of human leptomeningeal arteries in relation to perivascular drainage pathways in cerebral amyloid angiopathy”. Biochim Biophys Acta. 1862 (5): 1037—1046. PMC 4827375 . PMID 26327684. doi:10.1016/j.bbadis.2015.08.024. :1037–1046.
Gorelick, P. B.; Scuteri, A.; Black, S. E.; Decarli, C.; Greenberg, S. M.; Iadecola, C.; Launer, L. J.; Laurent, S.; Lopez, O. L.; Nyenhuis, D.; Petersen, R. C.; Schneider, J. A.; Tzourio, C.; Arnett, D. K.; Bennett, D. A.; Chui, H. C.; Higashida, R. T.; Lindquist, R.; Nilsson, P. M.; Roman, G. C.; Sellke, F. W.; Seshadri, S. (2011). „Vascular contributions to cognitive impairment and dementia: A statement for healthcare professionals from the american heart association/American stroke association”. Stroke. 42 (9): 2672—713. PMC 3778669 . PMID 21778438. doi:10.1161/STR.0b013e3182299496.
Arvanitakis Z., Leurgans S.E., Wang Z., Wilson R.S., Bennett D.A., Schneider J.A. (2011). „Cerebral amyloid angiopathy pathology and cognitive domains in older persons”. Ann Neurol. 69 (2): 320—327. PMC 3228518 . PMID 21387377. doi:10.1002/ana.22112. :320–327.
Weller R.O., Djuanda E., Yow H.-Y., Carare R.O. (2008). „Lymphatic drainage of the brain and the pathophysiology of neurological disease”. Acta Neuropathol. 117 (1): 1—14. PMID 19002474. doi:10.1007/s00401-008-0457-0. :1.
Akiyama H., Kondo H., Arai T.; et al. (2004). „Expression of BRI, the normal precursor of the amyloid protein of familial British dementia, in human brain”. Acta Neuropathol. 107 (1): 53—58. PMID 14586629. doi:10.1007/s00401-003-0783-1. :53–58
Yamada M. (2000). „Cerebral amyloid angiopathy: an overview”. Neuropathology. 20 (1): 8—22. PMID 10935432. doi:10.1046/j.1440-1789.2000.00268.x. :8–22.
Greenberg S.M., Gurol M.E., Rosand J., Smith E.E. (2004). „Amyloid angiopathy–related vascular cognitive impairment”. Stroke. 35 (11_suppl_1): 2616—2619. PMID 15459438. doi:10.1161/01.STR.0000143224.36527.44. :2616–2619.
Gilbert J.J., Vinters H.V. . Cerebral amyloid angiopathy: incidence and complications in the aging brain. I (1983). „Cerebral hemorrhage”. Stroke. 14 (6): 915—923. PMID 6658995. doi:10.1161/01.STR.14.6.915. .
Biffi A., Halpin A., Towfighi A.; et al. (2010). „Aspirin and recurrent intracerebral hemorrhage in cerebral amyloid angiopathy”. Neurology. 75 (8): 693—698. PMC 2931649 . PMID 20733144. doi:10.1212/WNL.0b013e3181eee40f. :693–698.
Zannis V.I., Breslow J.L., Utermann G.; et al. (1982). „Proposed nomenclature of apoE isoproteins, apoE genotypes, and phenotypes”. J Lipid Res. 23 (6): 911—914. PMID 7130859. doi:10.1016/S0022-2275(20)38094-9. .
Verghese, Philip B; Castellano, Joseph M; Holtzman, David M (2011). „Apolipoprotein E in Alzheimer's disease and other neurological disorders”. The Lancet Neurology. 10 (3): 241—252. ISSN 1474-4422. PMC 3132088 . PMID 21349439. doi:10.1016/s1474-4422(10)70325-2.
O'Donnell, H. C.; Rosand, J.; Knudsen, K. A.; Furie, K. L.; Segal, A. Z.; Chiu, R. I.; Ikeda, D.; Greenberg, S. M. (2000). „Apolipoprotein e genotype and the risk of recurrent lobar intracerebral hemorrhage”. N Engl J Med. 342 (4): 240—245. PMID 10648765. doi:10.1056/NEJM200001273420403.
Graff-Radford J., Madhavan M., Vemuri P.; et al. (2016). „Atrial fibrillation, cognitive impairment, and neuroimaging”. Alzheimers Dement. 12 (4): 391—398. PMC 4841716 . PMID 26607820. doi:10.1016/j.jalz.2015.08.164. :391–398.
Mittal, S.; Wu, Z.; Neelavalli, J.; Haacke, E. M. (2009). „Susceptibility-Weighted Imaging: Technical Aspects and Clinical Applications, Part 2”. American Journal of Neuroradiology. 30 (2): 232—252. ISSN 0195-6108. PMC 3805391 . PMID 19131406. doi:10.3174/ajnr.A1461.

harvard.edu

adsabs.harvard.edu

Roher A.E., Lowenson J.D., Clarke S.; et al. (1993). „beta-Amyloid-(1-42) is a major component of cerebrovascular amyloid deposits: implications for the pathology of Alzheimer disease”. Proc Natl Acad Sci U S A. 90 (22): 10836—10840. Bibcode:1993PNAS...9010836R. PMC 47873 . PMID 8248178. doi:10.1073/pnas.90.22.10836 . :10836–10840.

nih.gov

ncbi.nlm.nih.gov

Greenberg, S. M.; Charidimou, A. (2018). „Diagnosis of cerebral amyloid angiopathy: evolution of the Boston criteria”. Stroke. 49 (2): 491—497. PMC 5892842 . PMID 29335334. doi:10.1161/STROKEAHA.117.016990.
Revesz T., Holton J.L., Lashley T.; et al. (2009). „Genetics and molecular pathogenesis of sporadic and hereditary cerebral amyloid angiopathies”. Acta Neuropathol. 118 (1): 115—130. PMC 2844092 . PMID 19225789. doi:10.1007/s00401-009-0501-8. :115–130.
Roher A.E., Lowenson J.D., Clarke S.; et al. (1993). „beta-Amyloid-(1-42) is a major component of cerebrovascular amyloid deposits: implications for the pathology of Alzheimer disease”. Proc Natl Acad Sci U S A. 90 (22): 10836—10840. Bibcode:1993PNAS...9010836R. PMC 47873 . PMID 8248178. doi:10.1073/pnas.90.22.10836 . :10836–10840.
Attems J., Lintner F., Jellinger K.A. (2004). „Amyloid β peptide 1–42 highly correlates with capillary cerebral amyloid angiopathy and Alzheimer disease pathology”. Acta Neuropathol. 107 (5): 479—480. PMID 14986026. doi:10.1007/s00401-004-0866-7. :283–291.
Keable A., Fenna K., Yuen H.M.; et al. (2016). „Deposition of amyloid β in the walls of human leptomeningeal arteries in relation to perivascular drainage pathways in cerebral amyloid angiopathy”. Biochim Biophys Acta. 1862 (5): 1037—1046. PMC 4827375 . PMID 26327684. doi:10.1016/j.bbadis.2015.08.024. :1037–1046.
Gorelick, P. B.; Scuteri, A.; Black, S. E.; Decarli, C.; Greenberg, S. M.; Iadecola, C.; Launer, L. J.; Laurent, S.; Lopez, O. L.; Nyenhuis, D.; Petersen, R. C.; Schneider, J. A.; Tzourio, C.; Arnett, D. K.; Bennett, D. A.; Chui, H. C.; Higashida, R. T.; Lindquist, R.; Nilsson, P. M.; Roman, G. C.; Sellke, F. W.; Seshadri, S. (2011). „Vascular contributions to cognitive impairment and dementia: A statement for healthcare professionals from the american heart association/American stroke association”. Stroke. 42 (9): 2672—713. PMC 3778669 . PMID 21778438. doi:10.1161/STR.0b013e3182299496.
Arvanitakis Z., Leurgans S.E., Wang Z., Wilson R.S., Bennett D.A., Schneider J.A. (2011). „Cerebral amyloid angiopathy pathology and cognitive domains in older persons”. Ann Neurol. 69 (2): 320—327. PMC 3228518 . PMID 21387377. doi:10.1002/ana.22112. :320–327.
Weller R.O., Djuanda E., Yow H.-Y., Carare R.O. (2008). „Lymphatic drainage of the brain and the pathophysiology of neurological disease”. Acta Neuropathol. 117 (1): 1—14. PMID 19002474. doi:10.1007/s00401-008-0457-0. :1.
Akiyama H., Kondo H., Arai T.; et al. (2004). „Expression of BRI, the normal precursor of the amyloid protein of familial British dementia, in human brain”. Acta Neuropathol. 107 (1): 53—58. PMID 14586629. doi:10.1007/s00401-003-0783-1. :53–58
Yamada M. (2000). „Cerebral amyloid angiopathy: an overview”. Neuropathology. 20 (1): 8—22. PMID 10935432. doi:10.1046/j.1440-1789.2000.00268.x. :8–22.
Greenberg S.M., Gurol M.E., Rosand J., Smith E.E. (2004). „Amyloid angiopathy–related vascular cognitive impairment”. Stroke. 35 (11_suppl_1): 2616—2619. PMID 15459438. doi:10.1161/01.STR.0000143224.36527.44. :2616–2619.
Gilbert J.J., Vinters H.V. . Cerebral amyloid angiopathy: incidence and complications in the aging brain. I (1983). „Cerebral hemorrhage”. Stroke. 14 (6): 915—923. PMID 6658995. doi:10.1161/01.STR.14.6.915. .
Biffi A., Halpin A., Towfighi A.; et al. (2010). „Aspirin and recurrent intracerebral hemorrhage in cerebral amyloid angiopathy”. Neurology. 75 (8): 693—698. PMC 2931649 . PMID 20733144. doi:10.1212/WNL.0b013e3181eee40f. :693–698.
Zannis V.I., Breslow J.L., Utermann G.; et al. (1982). „Proposed nomenclature of apoE isoproteins, apoE genotypes, and phenotypes”. J Lipid Res. 23 (6): 911—914. PMID 7130859. doi:10.1016/S0022-2275(20)38094-9. .
Verghese, Philip B; Castellano, Joseph M; Holtzman, David M (2011). „Apolipoprotein E in Alzheimer's disease and other neurological disorders”. The Lancet Neurology. 10 (3): 241—252. ISSN 1474-4422. PMC 3132088 . PMID 21349439. doi:10.1016/s1474-4422(10)70325-2.
O'Donnell, H. C.; Rosand, J.; Knudsen, K. A.; Furie, K. L.; Segal, A. Z.; Chiu, R. I.; Ikeda, D.; Greenberg, S. M. (2000). „Apolipoprotein e genotype and the risk of recurrent lobar intracerebral hemorrhage”. N Engl J Med. 342 (4): 240—245. PMID 10648765. doi:10.1056/NEJM200001273420403.
Graff-Radford J., Madhavan M., Vemuri P.; et al. (2016). „Atrial fibrillation, cognitive impairment, and neuroimaging”. Alzheimers Dement. 12 (4): 391—398. PMC 4841716 . PMID 26607820. doi:10.1016/j.jalz.2015.08.164. :391–398.
Mittal, S.; Wu, Z.; Neelavalli, J.; Haacke, E. M. (2009). „Susceptibility-Weighted Imaging: Technical Aspects and Clinical Applications, Part 2”. American Journal of Neuroradiology. 30 (2): 232—252. ISSN 0195-6108. PMC 3805391 . PMID 19131406. doi:10.3174/ajnr.A1461.

worldcat.org

Verghese, Philip B; Castellano, Joseph M; Holtzman, David M (2011). „Apolipoprotein E in Alzheimer's disease and other neurological disorders”. The Lancet Neurology. 10 (3): 241—252. ISSN 1474-4422. PMC 3132088 . PMID 21349439. doi:10.1016/s1474-4422(10)70325-2.
Mittal, S.; Wu, Z.; Neelavalli, J.; Haacke, E. M. (2009). „Susceptibility-Weighted Imaging: Technical Aspects and Clinical Applications, Part 2”. American Journal of Neuroradiology. 30 (2): 232—252. ISSN 0195-6108. PMC 3805391 . PMID 19131406. doi:10.3174/ajnr.A1461.