Foreman(2006).”Attention deficit hyperactivity disorder: legal and ethical aspects”(på engelska).Archives of Disease in Childhood91(2): sid.192–194. doi:10.1136/adc.2004.064576.
Lange et al.(2010).”The history of attention deficit hyperactivity disorder”(på engelska).Attention Deficit and Hyperactivity Disorders2(4): sid.241–255. doi:10.1007/s12402-010-0045-8. PMID 21258430.
Faraone S. V, Buitelaar J(10 september 2009).”Comparing the efficacy of stimulants for ADHD in children and adolescents using meta-analysis”.Eur Child Adolesc Psychiatry19(4): sid.353–364. doi:10.1007/s00787-009-0054-3. PMID 19763664.
Arnsten A. F. T(19 juli 2006).”Stimulants: Therapeutic Actions in ADHD”.Neuropsychopharmacology31: sid.2376–2383. doi:10.1038/sj.npp.1301164. PMID 16855530.
Faraone, Stephen V.; Larsson, Henrik(2019).”Genetics of attention deficit hyperactivity disorder”.Molecular Psychiatry24(4): sid.562–575. doi:10.1038/s41380-018-0070-0. ISSN1476-5578. PMID 29892054. PMC: 6477889. https://pubmed.ncbi.nlm.nih.gov/29892054.Läst 23 april 2024. ”The mean heritability across 37 twin studies of ADHD or measures of inattentiveness and hyperactivity is 74% (Fig. 1). A similar heritability estimate of around 80% was seen in a study of MZ and DZ twins, full siblings, and maternal and paternal half-siblings [5]. The heritability is similar in males and females and for the inattentive and hyperactive-impulsive components of ADHD [6–8].”.
Glover V(April 2011).”Annual Research Review: Prenatal stress and the origins of psychopathology: an evolutionary perspective”.J Child Psychol Psychiatry52(4): sid.356–67. doi:10.1111/j.1469-7610.2011.02371.x. PMID 21250994.
Mayes R, Bagwell C, Erkulwater J(2008).”ADHD and the rise in stimulant use among children”.Harv Rev Psychiatry16(3): sid.151–166. doi:10.1080/10673220802167782. PMID 18569037.
”Skrivs damp och adhd med små eller stora bokstäver? – Frågelådan i svenska”.Språkrådet. Institutet för språk och folkminnen (Isof). https://frageladan.isof.se/visasvar.py?svar=48228.Läst 21 april 2024. ”Adhd är en förkortning av attention deficit hyperactivity disorder, ungefär ’uppmärksamhetsstörning med hyperaktivitetssyndrom’. Vi rekommenderar små bokstäver för damp och adhd. Båda orden är ursprungligen förkortningar av engelska uttryck. Sådana förkortningsord skrivs oftast med stora bokstäver så länge de är nya men med små bokstäver enligt svenska skrivregler när de blivit vanliga i allmänspråket, vilket damp och adhd har blivit. I medicinska texter, framför allt i vetenskaplig litteratur och i journaltext, behålls dock ibland skrivsättet med versaler: DAMP och ADHD.”
Lange et al.(2010).”The history of attention deficit hyperactivity disorder”(på engelska).Attention Deficit and Hyperactivity Disorders2(4): sid.241–255. doi:10.1007/s12402-010-0045-8. PMID 21258430.
Faraone S. V, Buitelaar J(10 september 2009).”Comparing the efficacy of stimulants for ADHD in children and adolescents using meta-analysis”.Eur Child Adolesc Psychiatry19(4): sid.353–364. doi:10.1007/s00787-009-0054-3. PMID 19763664.
Arnsten A. F. T(19 juli 2006).”Stimulants: Therapeutic Actions in ADHD”.Neuropsychopharmacology31: sid.2376–2383. doi:10.1038/sj.npp.1301164. PMID 16855530.
Faraone, Stephen V.; Larsson, Henrik(2019).”Genetics of attention deficit hyperactivity disorder”.Molecular Psychiatry24(4): sid.562–575. doi:10.1038/s41380-018-0070-0. ISSN1476-5578. PMID 29892054. PMC: 6477889. https://pubmed.ncbi.nlm.nih.gov/29892054.Läst 23 april 2024. ”The mean heritability across 37 twin studies of ADHD or measures of inattentiveness and hyperactivity is 74% (Fig. 1). A similar heritability estimate of around 80% was seen in a study of MZ and DZ twins, full siblings, and maternal and paternal half-siblings [5]. The heritability is similar in males and females and for the inattentive and hyperactive-impulsive components of ADHD [6–8].”.
Glover V(April 2011).”Annual Research Review: Prenatal stress and the origins of psychopathology: an evolutionary perspective”.J Child Psychol Psychiatry52(4): sid.356–67. doi:10.1111/j.1469-7610.2011.02371.x. PMID 21250994.
Mayes R, Bagwell C, Erkulwater J(2008).”ADHD and the rise in stimulant use among children”.Harv Rev Psychiatry16(3): sid.151–166. doi:10.1080/10673220802167782. PMID 18569037.
Faraone, Stephen V.; Larsson, Henrik(2019).”Genetics of attention deficit hyperactivity disorder”.Molecular Psychiatry24(4): sid.562–575. doi:10.1038/s41380-018-0070-0. ISSN1476-5578. PMID 29892054. PMC: 6477889. https://pubmed.ncbi.nlm.nih.gov/29892054.Läst 23 april 2024. ”The mean heritability across 37 twin studies of ADHD or measures of inattentiveness and hyperactivity is 74% (Fig. 1). A similar heritability estimate of around 80% was seen in a study of MZ and DZ twins, full siblings, and maternal and paternal half-siblings [5]. The heritability is similar in males and females and for the inattentive and hyperactive-impulsive components of ADHD [6–8].”.
Handbook of medical sociology(6th ed). Vanderbilt University Press. 2010. ISBN 978-0-8265-1722-7. OCLC719387793. https://www.worldcat.org/oclc/719387793.Läst 5 maj 2021. ”The hypothesis that ADHD is caused by dopamine deficiency is derived from post hoc pharmaceutical intervention, because Ritalin has been observed to help some children with ADHD while it is simultaneously believed to increase dopamine levels in the brain. However, direct measurement of dopamine levels in the brain cannot be sampled from living people, so they have to be inferred from dopamine metabolites in the blood, urine, or cerebrospinal fluid. The validity of such mea-surement is questionable given the existence of dopamine in other parts of the body. For similar reasons, the setting of normal levels of dopamine in the brain, from which people diagnosed with ADHD are supposed to deviate, is also problematic (Yuwiler, Brammer, and Yuwiler 1994). Indeed, it is quite possible that Ritalin’s effects on some ADHD children are via some mecha-nism other than dopamine increase (Glenmullen 2000). Thus, the biomedicalism claim that rising pharmaceuticalization, with respect to ADHD, is due to increased identification of people with bio-logical markers of the disease is not convincing.”
Svenaeus, Fredrik(2013).Homo Patologicus: medicinska diagnoser i vår tid. Tankekraft. sid.96-97. ISBN 978-91-86273-40-8. OCLC940189153. http://worldcat.org/oclc/940189153.Läst 5 maj 2021. ”För många fungerar nog medicineringen som ett bevis på att det är något biologiskt och sjukdomslikt som ligger bakom, men som jag redan varit inne på i kapitlet om ångest tror jag vi ska vara försiktiga med att dra sådana slutsatser. Förmodligen kommer vi i framtiden kunna påverka många personlighetsdrag med hjälp av kemiska substanser. I själva verket är det just det vi gör med hjälp av antidepressiva, ångesthämande och centralstimulerande medel när de har goda effekter på symtomen som definierar det vi kallar för psykiska störningar.”
Faraone, Stephen V.; Larsson, Henrik(2019).”Genetics of attention deficit hyperactivity disorder”.Molecular Psychiatry24(4): sid.562–575. doi:10.1038/s41380-018-0070-0. ISSN1476-5578. PMID 29892054. PMC: 6477889. https://pubmed.ncbi.nlm.nih.gov/29892054.Läst 23 april 2024. ”The mean heritability across 37 twin studies of ADHD or measures of inattentiveness and hyperactivity is 74% (Fig. 1). A similar heritability estimate of around 80% was seen in a study of MZ and DZ twins, full siblings, and maternal and paternal half-siblings [5]. The heritability is similar in males and females and for the inattentive and hyperactive-impulsive components of ADHD [6–8].”.
