Guthrie, FJ (2000). "Food sources of added sweeteners in the diets of Americans". Journals of American Dietetic Association. Cilt 100. ss. 43-51. doi:10.1016/S0002-8223(00)00018-3.
Beyer, PL (2005). "Fructose intake at current levels in the United States may cause gastrointestinal distress in normal adults". J. Am. Diet. Assoc. 105 (10). ss. 1559-1566. doi:10.1016/j.jada.2005.07.002. PMID16183355.
Beyer, PL (2005). "Fructose intake at current levels in the United States may cause gastrointestinal distress in normal adults". J. Am. Diet. Assoc. 105 (10). ss. 1559-66. doi:10.1016/j.jada.2005.07.002. PMID16183355.
Jürgens H, Haass W, Castañeda TR; ve diğerleri. (2005). "Consuming fructose-sweetened beverages increases body adiposity in mice". Obes. Res. 13 (7). ss. 1146-56. doi:10.1038/oby.2005.136. PMID16076983.KB1 bakım: Diğerlerinin yanlış kullanımı (link) KB1 bakım: Birden fazla ad: yazar listesi (link)
McPherson, JD (Kasım 1988). "Role of fructose in glycation and cross-linking of proteins. PMID 3132203". Biochemistry. 27 (5). ss. 1901-7. doi:10.1021/bi00406a016.
Teff, KL (Haziran 2004). "Dietary fructose reduces circulating insulin and leptin, attenuates postprandial suppression of ghrelin, and increases triglycerides in women". J Clin Endocrinol Metab. 89 (6). ss. 2963-72. doi:10.1210/jc.2003-031855. PMID15181085.
Ouyang X, Cirillo P, Sautin Y; ve diğerleri. (Haziran 2008). "Fructose consumption as a risk factor for non-alcoholic fatty liver disease". J. Hepatol. 48 (6). ss. 993-9. doi:10.1016/j.jhep.2008.02.011. PMC2423467 $2. PMID18395287.KB1 bakım: Diğerlerinin yanlış kullanımı (link) KB1 bakım: Birden fazla ad: yazar listesi (link)
Ushijima, K (1991). "Absorption of fructose by isolated small intestine of rats is via a specific saturable carrier in the absence of glucose and by the disaccharidase-related transport system in the presence of glucose". Journal of Nurtition. 125 (8). ss. 2156-2164. PMID7643250.
Beyer, PL (2005). "Fructose intake at current levels in the United States may cause gastrointestinal distress in normal adults". J. Am. Diet. Assoc. 105 (10). ss. 1559-1566. doi:10.1016/j.jada.2005.07.002. PMID16183355.
Beyer, PL (2005). "Fructose intake at current levels in the United States may cause gastrointestinal distress in normal adults". J. Am. Diet. Assoc. 105 (10). ss. 1559-66. doi:10.1016/j.jada.2005.07.002. PMID16183355.
Parniak, MA; Kalant, N (1988). "Enhancement of glycogen concentrations in primary cultures of rat hepatocytes exposed to glucose and fructose". Biochemical Journal. 251 (3). ss. 795-802. PMC1149073 $2. PMID3415647.
Havel PJ (2001). "Peripheral signals conveying metabolic information to the brain: short-term and long-term regulation of food intake and energy homeostasis". Exp. Biol. Med. (Maywood). 226 (11). ss. 963-77. PMID11743131.
Jürgens H, Haass W, Castañeda TR; ve diğerleri. (2005). "Consuming fructose-sweetened beverages increases body adiposity in mice". Obes. Res. 13 (7). ss. 1146-56. doi:10.1038/oby.2005.136. PMID16076983.KB1 bakım: Diğerlerinin yanlış kullanımı (link) KB1 bakım: Birden fazla ad: yazar listesi (link)
Teff, KL (Haziran 2004). "Dietary fructose reduces circulating insulin and leptin, attenuates postprandial suppression of ghrelin, and increases triglycerides in women". J Clin Endocrinol Metab. 89 (6). ss. 2963-72. doi:10.1210/jc.2003-031855. PMID15181085.
Ouyang X, Cirillo P, Sautin Y; ve diğerleri. (Haziran 2008). "Fructose consumption as a risk factor for non-alcoholic fatty liver disease". J. Hepatol. 48 (6). ss. 993-9. doi:10.1016/j.jhep.2008.02.011. PMC2423467 $2. PMID18395287.KB1 bakım: Diğerlerinin yanlış kullanımı (link) KB1 bakım: Birden fazla ad: yazar listesi (link)